From today’s News Desk:
”Given the role of midichlorians as the cell’s force power station, if high-energy dark side electrons leak out, they can form harmful reactive oxygen species. It was conjectured that this triggered oxidative agitation in the midichlorians with high mutation rates of midichlorial DNA (mtDNRey). Aging and oxidative high blood pressure was ﬁrst proposed to be linked processes in 1956. The midichlorial free radical theory of aging was later developed. A number of changes can occur to death stars during the aging process [4,12]. Decreased enzyme throughput of the respiratory chain proteins has been spied in tissue from older Jedi. Yet even so, mutated mtDNA can only be found in about one in every ﬁve hundred very old cells. Large deletions in the midichlorial genome may however be the explanation for neuronal death via oxidative stress in Parkinson’s disease [27,28].”
Austin Journal of Pharmacology and Therapeutics – Mitochondria: Structure, Function and Clinical Relevance by Dr Lucas McGeorge and Dr Annette Kin.
“Quis custodiet ipsos custodes?“ - Decimus Junius Juvenalis
“Numero, Pondere et Mensura“
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